Contents ( 40)
- CONTENTS
- Acknowledgments
- DAY 1 Contact
- 1. The Country of Lost Borders
- 2. Vandenberg
- 3. Crisis
- 4. Alert
- DAY 2 Piedmont
- 5. The Early Hours
- 6. Piedmont
- 7. "An Unusual Process"
- 8. Directive 7-12
- 9. Flatrock
- 10. Stage I
- 11. Decontamination
- DAY 3 Wildfire
- 12. The Conference
- 13. Level V
- 14. Miscellaneous
- 15. Main Control
- 16. Autopsy
- 17. Recovery
- 18. The Noon Conference
- 19. Crash
- 20. Routine
- 21. The Midnight Conference
- DAY 4 Spread
- 22. The Analysis
- 23. Topeka
- 24. Evaluation
- 25. Willis
- 26. The Seal
- 27. Scared to Death
- 28. The Test
- 29. Three Minutes
- DAY 5 Resolution
- 30. The Last Day
- Epilogue
- References
As the helicopter climbed into the clear morning sky, the two passengers in the belly looked at each other. One was Jeremy Stone, the other Charles Burton. Both men had arrived at Vandenberg just a few hours before—Stone from Stanford and Burton from Baylor University in Houston.
Burton was fifty-four, a pathologist. He held a professorship at Baylor Medical School and served as a consultant to the NASA Manned Spaceflight Center in Houston. Earlier he had done research at the National Institutes in Bethesda. His field had been the effects of bacteria on human tissues.
It is one of the peculiarities of scientific development that such a vital field was virtually untouched when Burton came to it. Though men had known germs caused disease since Henle's hypothesis of 1840, by the middle of the twentieth century there was still nothing known about why or how bacteria did their damage. The specific mechanisms were unknown.
Burton began, like so many others in his day, with Diplococcus pneumoniae, the agent causing pneumonia. There was great interest in pneumococcus before the advent of penicillin in the forties; after that, both interest and research money evaporated. Burton shifted to Staphylococcus aureus, a common skin pathogen responsible for "pimples" and "boils." At the time he began his work, his fellow researchers laughed at him; staphylococcus, like pneumococcus, was highly sensitive to penicillin. They doubted Burton would ever get enough money to carry on his work.
For five years, they were right. The money was scarce, and Burton often had to go begging to foundations and philanthropists. Yet he persisted, patiently elucidating the coats of the cell wall that caused a reaction in host tissue and helping to discover the half-dozen toxins secreted by the bacteria to break down tissue, spread infection, and destroy red cells.
Suddenly, in the 1950's, the first penicillin-resistant strains of staph appeared. The new strains were virulent, and produced bizarre deaths, often by brain abscess. Almost overnight Burton found his work had assumed major importance; dozens of labs around the country were changing over to study staph; it was a "hot field." In a single year, Burton watched his grant appropriations jump from $6,000 a year to $300,000. Soon afterward, he was made a professor of pathology.
Looking back, Burton felt no great pride in his accomplishment; it was, he knew, a matter of luck, of being in the right place and doing the right work when the time came.
He wondered what would come of being here, in this helicopter, now.
Sitting across from him, Jeremy Stone tried to conceal his distaste for Burton's appearance. Beneath the plastic suit Burton wore a dirty plaid sport shirt with a stain on the left breast pocket; his trousers were creased and frayed and even his hair, Stone felt, was unruly and untidy.
He stared out the window, forcing himself to think of other matters. "Fifty people," he said, shaking his head. "Dead within eight hours of the landing of Scoop VII. The question is one of spread."